Fig. 2
From: Protective mechanism of quercetin and its derivatives in viral-induced respiratory illnesses
Illustration of influenza virus life cycle and the pathways modulated by quercetin. Influenza virus utilizes hemagglutinin (HA) receptor for binding and entry. Moreover, membrane-2 (M2) ion channel promotes fusion and uncoating, and neuraminidase (NA) facilitates the progeny virus release. Collectively, quercetin interferes at different steps including viral binding, uncoating, mRNA synthesis, negative-strand synthesis, assembly, and release [12]