Microbiological analysis of nosocomial pneumonia at Tanta University Chest Hospital

The Egyptian Journal of Bronchology

Table 2 Clinical data of patients in HAP and VAP groups

Laboratory investigations		Group						T-test
Laboratory investigations		HAP N = 50			VAP N = 50			t	p-value
TLC at time of NP diagnosis	Range	4.8	-	33	5	-	34	− 0.706	0.482
TLC at time of NP diagnosis	Mean ± SD	17.812	±	6.879	18.748	±	6.359	− 0.706	0.482
CRP at time of NP diagnosis	Range	12	-	210	60	-	200	− 4.106	< 0.001*
CRP at time of NP diagnosis	Mean ± SD	80.180	±	45.947	114.300	±	36.628	− 4.106	< 0.001*
D-dimer at time of NP diagnosis	Range	0.1	-	1.84	0.4	-	2.5	− 4.943	< 0.001*
D-dimer at time of NP diagnosis	Mean ± SD	0.662	±	0.376	1.134	±	0.561	− 4.943	< 0.001*
Chi-square		N		%	N		%	Χ²	P-value
Cause of admission	AE. COPD	18		36.00	18		36.00	25.083	0.009*
	AE. asthma	15		30.00	4		8.00
	AE. IPF	1		2.00	6		12.00
	AE. HP	4		8.00	3		6.00
	AE bronchiectasis	3		6.00	6		12.00
	AE. RA-ILD	1		2.00	0		0.00
	Obesity hypoventilation syndrome	3		6.00	6		12.00
	Organophosphorus poisoning	0		0.00	3		6.00
	Pleural effusion	3		6.00	0		0.00
	Pulmonary edema	0		0.00	3		6.00
	Lung cancer	2		4.00	0		0.00
	Acute upper airway obstruction	0		0.00	1		2.00
Onset of nosocomial pneumonia	Early	21		42.00	22		44.00	0.041	0.840
Onset of nosocomial pneumonia	Late	29		58.00	28		56.00	0.041	0.840
Radiological findings	Lobar pneumonia with pleural effusion	2		4.00	5		10.00	10.190	0.037*
	Lobar pneumonia	20		40.00	17		34.00
	Bronchopneumonia	17		34.00	26		52.00
	Lung abscess	7		14.00	2		4.00
	Bilateral GGO	4		8.00	0		0.00

AE. COPD acute exacerbation of chronic obstructive pulmonary diseases, AE. HP acute exacerbation of hypersensitivity pneumonitis, AE.IPF acute exacerbation of idiopathic pulmonary fibrosis, AE. RA-ILD acute exacerbation of rheumatoid arthritis-interstitial lung diseases, CRP C-reactive protein, GGO ground-glass opacities, NP nosocomial pneumonia, TLC total leukocytic count
*P-value is significant if < 0.05